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Background: Neuronal Damage Caused by Amphetamines Amphetamine and methamphetamine (METH) are psychomotor stimulants, causing increased arousal, sympathetic activation, reduced food intake, and hyperactivity following low-dose administration. In addition, these drugs exert a reinforcing effect following their self-administration and are, therefore, prone to repeated use and abuse. , 2005).

J. Control. Release 98, 337–353. Zhdanova, I. , and Giorgetti, M. (2002). Melatonin alters behavior and cAMP levels in nucleus accumbens induced by cocaine treatment. Brain Res. 956, 323–331. Zisapel, N. (2001). Melatonin-dopamine interactions: From basic neurochemistry to a clinical setting. Cell. Mol. Neurobiol. 21, 605–616. DOPAMINE-INDUCED BEHAVIORAL CHANGES AND OXIDATIVE STRESS IN METHAMPHETAMINE-INDUCED NEUROTOXICITY Taizo Kita,* Ikuko Miyazaki,y Masato Asanuma,y Mika Takeshima,* and George C.

1992). Periodicity of presentations of drugs of abuse and overdose in an emergency department. Clin. Toxicol. 30, 467–478. Reppert, S. , and Weaver, D. R. (2002). Coordination of circadian timing in mammals. Nature 418, 935–941. Roberts, D. , and Lynch, W. J. (2002). Patterns of cocaine self-administration in rats produced by various access conditions under a discrete trials procedure. Drug Alcohol Depend. 67, 291–299. Satel, S. , and Gawin, F. H. (1989). Seasonal cocaine abuse. Am. J. Psychiatry 146, 534–535.

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